[1] Zhao YX, Liu YZ, Zhou HY, et al. Intelligent gateway based human cardiopulmonary health monitoring system. J Sens, 2023; 2023, 3534224.
[2] Roth GA, Mensah GA, Johnson CO, et al. Global burden of cardiovascular diseases and risk factors, 1990-2019: update from the GBD 2019 study. J Am Coll Cardiol, 2020; 76, 2982−3021.
[3] GBD Chronic Respiratory Disease Collaborators. Prevalence and attributable health burden of chronic respiratory diseases, 1990-2017: a systematic analysis for the Global Burden of Disease Study 2017. Lancet Respir Med, 2020; 8, 585−96.
[4] Wan EYF, Fung WT, Schooling CM, et al. Blood pressure and risk of cardiovascular disease in UK biobank: a Mendelian randomization study. Hypertension, 2021; 77, 367−75.
[5] Chen SF, Lu XF. The impact of environmental factors on cardiopulmonary health. Biomed Environ Sci, 2024; 37, 1349−51.
[6]
[7] Holme JA, Brinchmann BC, Refsnes M, et al. Potential role of polycyclic aromatic hydrocarbons as mediators of cardiovascular effects from combustion particles. Environ Health, 2019; 18, 74.
[8] Olsson AC, Fevotte J, Fletcher T, et al. Occupational exposure to polycyclic aromatic hydrocarbons and lung cancer risk: a multicenter study in Europe. Occup Environ Med, 2010; 67, 98−103.
[9] Wu LH, Zhang XM, Wang F, et al. Occurrence of bisphenol S in the environment and implications for human exposure: a short review. Sci Total Environ, 2018; 615, 87−98.
[10] Li JJ, Wang GH. Airborne particulate endocrine disrupting compounds in China: compositions, size distributions and seasonal variations of phthalate esters and bisphenol A. Atmos Res, 2015; 154, 138−45.
[11] Heudorf U, Mersch-Sundermann V, Angerer J. Phthalates: toxicology and exposure. Int J Hyg Environ Health, 2007; 210, 623−34.
[12] Wang W, Leung AOW, Chu LH, et al. Phthalates contamination in China: status, trends and human exposure-with an emphasis on oral intake. Environ Pollut, 2018; 238, 771−82.
[13] Matsumoto M, Hirata-Koizumi M, Ema M. Potential adverse effects of phthalic acid esters on human health: a review of recent studies on reproduction. Regul Toxicol Pharmacol, 2008; 50, 37−49.
[14] Kashyap D, Agarwal T. Concentration and factors affecting the distribution of phthalates in the air and dust: a global scenario. Sci Total Environ, 2018; 635, 817−27.
[15] Janjua NR, Mortensen GK, Andersson AM, et al. Systemic uptake of diethyl phthalate, dibutyl phthalate, and butyl paraben following whole-body topical application and reproductive and thyroid hormone levels in humans. Environ Sci Technol, 2007; 41, 5564−70.
[16] Liang JF, Liu QS, Ren ZH, et al. Studying paraben-induced estrogen receptor- and steroid hormone-related endocrine disruption effects via multi-level approaches. Sci Total Environ, 2023; 869, 161793.
[17] Miller MR. Oxidative stress and the cardiovascular effects of air pollution. Free Radic Biol Med, 2020; 151, 69−87.
[18] Bae S, Pan XC, Kim SY, et al. Exposures to particulate matter and polycyclic aromatic hydrocarbons and oxidative stress in schoolchildren. Environ Health Perspect, 2010; 118, 579−83.
[19] Zhu XL, Zhang QL, Du XH, et al. Respiratory effects of traffic-related air pollution: a randomized, crossover analysis of lung function, airway metabolome, and biomarkers of airway injury. Environ Health Perspect, 2023; 131, 057002.
[20] Caramori G, Papi A. Oxidants and asthma. Thorax, 2004; 59, 170−3.
[21] Zhang YJ, Huang C, Lv YS, et al. Polycyclic aromatic hydrocarbon exposure, oxidative potential in dust, and their relationships to oxidative stress in human body: a case study in the indoor environment of Guangzhou, South China. Environ Int, 2021; 149, 106405.
[22] Lu SY, Li YX, Zhang JQ, et al. Associations between polycyclic aromatic hydrocarbon (PAH) exposure and oxidative stress in people living near e-waste recycling facilities in China. Environ Int, 2016; 94, 161−9.
[23] Kuang D, Zhang WZ, Deng QF, et al. Dose-response relationships of polycyclic aromatic hydrocarbons exposure and oxidative damage to DNA and lipid in coke oven workers. Environ Sci Technol, 2013; 47, 7446−56.
[24] Shi JZ, Zhao Y, Xue LJ, et al. Urinary metabolites of polycyclic aromatic hydrocarbons after short-term fine particulate matter exposure: a randomized crossover trial of air filtration. Environ Pollut, 2021; 285, 117258.
[25] Carrico C, Gennings C, Wheeler DC, et al. Characterization of weighted quantile sum regression for highly correlated data in a risk analysis setting. J Agric Biol Environ Stat, 2015; 20, 100−20.
[26] Tingley D, Yamamoto T, Hirose K. mediation: R package for causal mediation analysis. J Stat Softw, 2014; 59, 1−38.
[27] Li J, Zhu XY, Yu K, et al. Exposure to polycyclic aromatic hydrocarbons and accelerated DNA methylation aging. Environ Health Perspect, 2018; 126, 067005.
[28] Zhou Y, Liu YW, Sun HZ, et al. Associations of urinary polycyclic aromatic hydrocarbon metabolites with fractional exhaled nitric oxide and exhaled carbon monoxide: a cross-sectional study. Sci Total Environ, 2018; 618, 542−50.
[29] Gao CJ, Liu LY, Ma WL, et al. Bisphenol a in urine of Chinese young adults: concentrations and sources of exposure. Bull Environ Contam Toxicol, 2016; 96, 162−7.
[30] Miao Y, Chen PP, Zhang M, et al. Within-day variability, predictors, and risk assessments of exposure to parabens among Chinese adult men. Environ Res, 2023; 218, 115026.
[31] Wang H, Cui YX, Zhang F, et al. Association between urinary phthalate metabolites and hyperuricemia in US adults. Environ Sci Pollut Res, 2023; 30, 41445−59.
[32] Yin WJ, Hou J, Xu T, et al. Obesity mediated the association of exposure to polycyclic aromatic hydrocarbon with risk of cardiovascular events. Sci Total Environ, 2018; 616-617, 841-54.
[33] Alshaarawy O, Elbaz HA, Andrew ME. The association of urinary polycyclic aromatic hydrocarbon biomarkers and cardiovascular disease in the US population. Environ Int, 2016; 89-90, 174-8.
[34] Hu C, Hou J, Zhou Y, et al. Association of polycyclic aromatic hydrocarbons exposure with atherosclerotic cardiovascular disease risk: a role of mean platelet volume or club cell secretory protein. Environ Pollut, 2018; 233, 45−53.
[35] Jung JH, Hicken CE, Boyd D, et al. Geologically distinct crude oils cause a common cardiotoxicity syndrome in developing zebrafish. Chemosphere, 2013; 91, 1146−55.
[36] Dong YH, Chen L, Gao D, et al. Endogenous sex hormones homeostasis disruption combined with exogenous phthalates exposure increase the risks of childhood high blood pressure: a cohort study in China. Environ Int, 2022; 168, 107462.
[37] Chapman RS. Lung function and polycyclic aromatic hydrocarbons in China. Am J Respir Crit Care Med, 2016; 193, 814−5.
[38] Song XL, Wang C, He H, et al. Association of phthalate exposure with pulmonary function in adults: NHANES 2007-2012. Environ Res, 2023; 237, 116902.
[39] Beckman KB, Ames BN. Oxidative decay of DNA. J Biol Chem, 1997; 272, 19633−6.
[40] Graille M, Wild P, Sauvain JJ, et al. Urinary 8-OHdG as a biomarker for oxidative stress: a systematic literature review and meta-analysis. Int J Mol Sci, 2020; 21, 3743.
[41] Ferguson KK, Mcelrath TF, Pace GG, et al. Urinary polycyclic aromatic hydrocarbon metabolite associations with biomarkers of inflammation, angiogenesis, and oxidative stress in pregnant women. Environ Sci Technol, 2017; 51, 4652−60.
[42] Fattman CL, Schaefer LM, Oury TD. Extracellular superoxide dismutase in biology and medicine. Free Radic Biol Med, 2003; 35, 236−56.