[1] Fousteri G, SN Liossis, M Battaglia. Roles of the protein tyrosine phosphatase PTPN22 in immunity and autoimmunity. Clin Immunol, 2013; 149, 556−65. doi:  10.1016/j.clim.2013.10.006
[2] Krischer JP, X Liu, K Vehik, et al. Predicting Islet Cell Autoimmunity and Type 1 Diabetes: An 8-Year TEDDY Study Progress Report. Diabetes Care, 2019.
[3] Ferrante AW Jr. The immune cells in adipose tissue. Diabetes Obes Metab, 2013; 15(Suppl 3), 34−8.
[4] Yu X, JP Sun, Y He, et al. Structure, inhibitor, and regulatory mechanism of Lyp, a lymphoid-specific tyrosine phosphatase implicated in autoimmune diseases. Proc Natl Acad Sci USA, 2007; 104, 19767−72. doi:  10.1073/pnas.0706233104
[5] Wang Y, I Shaked, SM Stanford, et al. The autoimmunity-associated gene PTPN22 potentiates toll-like receptor-driven, type 1 interferon-dependent immunity. Immunity, 2013; 39, 111−22. doi:  10.1016/j.immuni.2013.06.013
[6] Clarke F, HA Purvis, C Sanchez-Blanco, et al. The protein tyrosine phosphatase PTPN22 negatively regulates presentation of immune complex derived antigens. Sci Rep, 2018; 8, 12692. doi:  10.1038/s41598-018-31179-x
[7] Li M, H Beauchemin, N Popovic, et al. The common, autoimmunity-predisposing 620Arg > Trp variant of PTPN22 modulates macrophage function and morphology. J Autoimmun, 2017.
[8] Salinas-Santander MA, RB Leon-Cachon, AC Cepeda-Nieto, et al. Assessment of biochemical parameters and characterization of alpha -308G/A and +1858C/T gene polymorphisms in the risk of obesity in adolescents. Biomed Rep, 2016; 4, 107−111. doi:  10.3892/br.2015.534
[9] Lumeng, CN, JL Bodzin, AR Saltiel. Obesity induces a phenotypic switch in adipose tissue macrophage polarization. J Clin Invest, 2007; 117, 175−84. doi:  10.1172/JCI29881
[10] Lumeng CN, JB DelProposto, DJ Westcott, et al. Phenotypic switching of adipose tissue macrophages with obesity is generated by spatiotemporal differences in macrophage subtypes. Diabetes, 2008; 57, 3239−46. doi:  10.2337/db08-0872