[1] Ning L, Rui X, Guorui L, et al. A novel mechanism for the protection against acute lung injury by melatonin: mitochondrial quality control of lung epithelial cells is preserved through SIRT3-dependent deacetylation of SOD2. Cell Mol Life Sci, 2022; 79, 610. doi:  10.1007/s00018-022-04628-0
[2] Li QW, Liao JZ, Chen WJ, et al. NAC alleviative ferroptosis in diabetic nephropathy via maintaining mitochondrial redox homeostasis through activating SIRT3-SOD2/Gpx4 pathway. Free Radic Biol Med, 2022; 187, 158−70. doi:  10.1016/j.freeradbiomed.2022.05.024
[3] Li SB, He YP, Chen KX, et al. RSL3 drives ferroptosis through NF-κB pathway activation and GPX4 depletion in glioblastoma. Oxid Med Cell Longev, 2021; 2021, 2915019. doi:  10.1155/2021/2915019
[4] Zheng DD, Liu J, Piao H, et al. ROS-triggered endothelial cell death mechanisms: focus on pyroptosis, parthanatos, and ferroptosis. Front Immunol, 2022; 13, 1039241. doi:  10.3389/fimmu.2022.1039241
[5] Zeng ZH, Yang YT, Dai XG, et al. Polydatin ameliorates injury to the small intestine induced by hemorrhagic shock via SIRT3 activation-mediated mitochondrial protection. Expert Opin Ther Targets, 2016; 20, 645−52. doi:  10.1080/14728222.2016.1177023
[6] Wu J, Deng ZY, Sun MM, et al. Polydatin protects against lipopolysaccharide-induced endothelial barrier disruption via SIRT3 activation. Lab Invest, 2020; 100, 643−56. doi:  10.1038/s41374-019-0332-8
[7] Arra M, Swarnkar G, Ke K, et al. LDHA-mediated ROS generation in chondrocytes is a potential therapeutic target for osteoarthritis. Nat Commun, 2020; 11, 3427. doi:  10.1038/s41467-020-17242-0
[8] Yao XM, Li W, Fang D, et al. Emerging roles of energy metabolism in ferroptosis regulation of tumor cells. Adv Sci (Weinh), 2021; 8, 2100997. doi:  10.1002/advs.202100997
[9] Yang J, Ma SY, Xu R, et al. Smart biomimetic metal organic frameworks based on ROS-ferroptosis-glycolysis regulation for enhanced tumor chemo-immunotherapy. J Control Release, 2021; 334, 21−33. doi:  10.1016/j.jconrel.2021.04.013