[1] Laina A, Stellos K, Stamatelopoulos K. Vascular ageing: Underlying mechanisms and clinical implications. Exp Gerontol, 2018; 109, 16−30. doi:  10.1016/j.exger.2017.06.007
[2] Yetim E, Topcuoglu MA, Yurur Kutlay N, et al. The association between telomere length and ischemic stroke risk and phenotype. Sci Rep, 2021; 11, 10967. doi:  10.1038/s41598-021-90435-9
[3] Zhang WL, Chen Y, Wang YY, et al. Short telomere length in blood leucocytes contributes to the presence of atherothrombotic stroke and haemorrhagic stroke and risk of post-stroke death. Clin Sci (Lond), 2013; 125, 27−36. doi:  10.1042/CS20120691
[4] Cao WJ, Li XG, Zhang XY, et al. No causal effect of telomere length on ischemic stroke and its subtypes: a Mendelian randomization study. Cells, 2019; 8, 159. doi:  10.3390/cells8020159
[5] Skrivankova VW, Richmond RC, Woolf BAR, et al. Strengthening the Reporting of Observational Studies in Epidemiology Using Mendelian Randomization: The STROBE-MR Statement. JAMA, 2021; 326, 1614−21. doi:  10.1001/jama.2021.18236
[6] Li C, Stoma S, Lotta LA, et al. Genome-wide association analysis in humans links nucleotide metabolism to leukocyte telomere length. Am J Hum Genet, 2020; 106, 389−404. doi:  10.1016/j.ajhg.2020.02.006
[7] Traylor M, Persyn E, Tomppo L, et al. Genetic basis of lacunar stroke: a pooled analysis of individual patient data and genome-wide association studies. Lancet Neurol, 2021; 20, 351−61. doi:  10.1016/S1474-4422(21)00031-4
[8] Liu Y, Liu SY, Xin JR, et al. Telomere length and hearing loss: a two-sample Mendelian randomization. Int J Environ Res Public Health, 2022; 19, 8937. doi:  10.3390/ijerph19158937
[9] Huang DQ, Lin SQ, He JT, et al. Association between COVID-19 and telomere length: a bidirectional Mendelian randomization study. J Med Virol, 2022; 94, 5345−53. doi:  10.1002/jmv.28008